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🌊 The Ocean Library · GP clinical topic

Obstructive Sleep Apnoea (OSA)

Written and reviewed by practising UK GPs, overseen by our Clinical Advisory Officer. Last updated 16 Dec 2025.

🧭 When to suspect

Suspect obstructive sleep apnoea (OSA) in a patient – classically an overweight, middle-aged man – who reports the cardinal triad of loud habitual snoring, witnessed apnoeas (a bed partner describing pauses, gasping or choking during sleep), and excessive daytime sleepiness that persists despite adequate time in bed. Supporting features include morning headache, poor concentration, low mood and nocturia.

It is worth holding two terms apart. OSA describes the breathing events found on a sleep study, whereas obstructive sleep apnoea/hypopnoea syndrome (OSAHS) – sometimes called OSA syndrome – means those events plus daytime symptoms, usually sleepiness. This distinction matters for driving advice, because Driver and Vehicle Licensing Agency (DVLA) duties turn on excessive sleepiness, not on the sleep-study finding alone.

The key primary-care skills are threefold: raise suspicion (including in patients who are not obese), screen and refer for an objective sleep study, and – above all – act first on the sleepy driver. Severity is graded by the apnoea–hypopnoea index (AHI), the number of apnoeas and hypopnoeas per hour of sleep.

AHI (events per hour) Classification Typical direction of treatment
< 5 Normal No OSAHS on this measure
5 to < 15 Mild OSAHS Lifestyle ± mandibular advancement splint; CPAP if symptomatic and first-line is insufficient
15 to < 30 Moderate OSAHS CPAP first-line, alongside lifestyle
≥ 30 Severe OSAHS CPAP first-line; ensure prompt treatment

Raise suspicion most strongly with obesity (especially central, with a large collar size), male sex, middle or older age, and post-menopausal status in women. Other contributors are upper-airway and craniofacial anatomy (retrognathia, micrognathia, macroglossia, enlarged tonsils), alcohol and sedatives, smoking, hypothyroidism, acromegaly and a family history. In children, the commonest cause is adenotonsillar hypertrophy; suspect it with habitual snoring, mouth breathing, restless sleep or behavioural and school difficulties.

Source: NICE NG202


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